After Antibiotics

How To Deal With Gastrointestinal Distress

After Antibiotics. Healthy Living Magazine

After Antibiotics. Healthy Living Magazine

If you’re one of the 80% of Americans who’ve been prescribed antibiotics, it’s not unlikely their use could be the cause of your chronic gastrointestinal discomfort, inflammation and other symptoms of stomach upset. These symptoms could also be the precursors to liver disease.

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Antibiotic use, even when necessary, leads to overgrowth of disease-causing bacteria. That is because antibiotics are rarely deadly to only one bacterium. They end up killing both the bad guys that are wanted dead in order to facilitate recovery yet also cause collateral damage to populations of friendly bacteria necessary for digestion, detoxification, immune cell production and utilization of nutrients. Very often after antibiotic use, the body undergoes an explosion in the population of the bad guys including the spore-forming bacterium Clostridium difficile, the causative agent of colitis (inflammation of the colon) and diarrhea that follows antibiotic intake.

This inflammatory condition, if left untreated, could result in further damage to other organs. Chronic lowgrade inflammation involves endotoxins derived from the pathogenic gut flora when they are overgrown from antibiotics. Interestingly, these gut-derived bacterial endotoxins have been considered to be an important cofactor that mediates the pathogenesis of liver injury in non-alcoholic steatohepatitis (NASH), also known as fatty liver disease.

after antibiotics

Bovine colostrum, the first milk derived immediately following the birth of the calf, is rich in immune and growth factors and has been found to have neutralizing activity against poisons produced by C. difficile. These poisons are called toxins A and B. This finding is important since toxinneutralizing activity in the human colon is needed to have therapeutic effects. The IgG family of immunoglobulins that kill these bacteria are the predominant ones in bovine colostrum (92%).

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Both human and animal studies have found protection from colostrum containing high concentrations of IgG. In fact, use of colostrum is cited by the University of Florida Medical School, in Gainesville, in online medical information as standard for this bacterium. Studies further validate that using colostrum protects against liver disease.

immune milk

Because of its growth and immune factors necessary to repair tissues and muscle growth and blood sugar control, first-milking colostrum is attractive to consume regularly as an anti-aging and cell renewal super food. It is especially indicated when one is being prescribed antibiotics.

First-milking colostrum is obtained from dairy cows within the first six hours and is what’s always left after the calf is finished. True colostrum is produced before the birth of the calf and can only be collected for a short period of time without being diluted by the subsequent production of milk. At the time of birth, potency is at its peak. The active elements such as immune factors, growth factors, antioxidants and anti-inflammatory agents are at their highest concentrations. However, in fewer than 12 hours, the concentration of these components is only half of what it was at the time of birth. What this means is that the sooner the colostrum is collected, the less diluted it is with milk and the greater the concentration of beneficial factors. People take colostrum capsules as supplements or as a meal in shakes and smoothies.

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ReferencesAdar T, Ben Ya’acov A, Lalazar G, Lichtenstein Y, Nahman D, Mizrahi M, Wong V, Muller B, Rawlin G, Ilan Y. Oral administration of immunoglobulin G-enhanced colostrum alleviates insulin resistance and liver injury and is associated with alterations in natural killer T cells. Clin Exp Immunol. 2012 Feb; 167(2): 252–260. doi: 10.1111/j.1365-2249.2011.04511.x PMCID: PMC 3278691 Kim K, et al. In vitro and in vivo neutralizing activity of human colostrum and milk against purified toxins A and B of Clostridium difficile. J Infectious Diseases, 1984;150(1):57-61. See for example: “http://216.239.39.100/search?q=cache:Gml7vqrkNwC:www.medinfo.ufl.edu/year2/mmid/bms5300/bugs/closdif.html +Clostridium+difficile+colostrum&hl=en&ie=UTF-8” Kudo H, Takahara T, Yata Y, Kawai K, Zhang W, Sugiyama T. Lipopolysaccharide triggered TNF-alpha-induced hepatocyte apoptosis in a murine non-alcoholic steatohepatitis model. J Hepatol. 2009;51:168–175. [PubMed] Verdam FJ, Rensen SS, Driessen A, Greve JW, Buurman WA. Novel evidence for chronic exposure to endotoxin in human nonalcoholic steatohepatitis. J Clin Gastroenterol. 2011;45:149–152. [PubMed]
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